Stephen Miller's Specific Immunity
Stephen Miller
Golden West College

Specific Immunity


SPECIFIC HOST DEFENSES

SPECIFIC HOST DEFENSES

Immune System: 2 Parts

  1) Humoral Immune System: Involves specific antibodies in the blood and lymph.

      (The body's humors)

         Produced by B cells.

  2) Cellular Immune System: Involves T cells.   That do not produce antibodies but secrete  cytokines.

 

Specialized lymphocytes respond to intracellular Ags

B cells mature in the bone marrow.

T cells mature in the thymus.

After maturation they migrate to lymphoid tissue.

 

Kinds of Immunity

Innate Immunity and Acquired Immunity

Acquired Immunity : Resistance to infection due to activity of antibodies.

            a) Active Immunity

            b) Passive Immunity

Naturally Acquired Active Immunity

1) Stimulus : Contact with a live microbe by natural processes. e.g. Infection; illness

2) Response : Symptoms of disease or subclinical RXN

     Active production of specific antibodies to the

     pathogen.

3) Duration : Long term (months - years),

     sometimes life long.

     Lots of exceptions: e.g. Influenza

Artificially Acquired Active Immunity

Antigen is intentionally introduced into the body.

1) Stimulus : Vaccines/Immunization:

     a) Killed pathogens (or their proteins alone)

     b) Attenuated/weakened live pathogens

     c) Inactivated toxins (toxoids)

2) Response : Production of specific antibodies without developing symptoms of disease (or prodromal symptoms only)

3) Duration : Variable (Months-years-life time)

 

Passive Immunity: Immunity acquired through transfer of antibodies.

Naturally Acquired Passive Immunity

1) Mother to fetus through the placenta  (placental transfer) or in colostrum/milk during nursing.

2) Response : No immune response. Acquisition of antibody only.

3) Duration : Short term (a few weeks -months)

 

Artificially Acquired Passive Immunity
1) Antibodies formed in one individual   transferred/injected into another individual.

    Immune serum/gamma globulin

2) Response : No immune response, acquisition of antibody only.

3) Duration : Very short (2-3 weeks) e.g. Venomous snake bites, Tetanus, Hep. A, Diphtheria, Botulism.

 

ANTIGENS AND ANTIBODIES
Specific immune response involves production of specific antibodies (Ab) against specific antigens (Ag).

The nature of antigens/Immunogens

1) Definition: An Immunogen is any  substance that when introduced into the body stimulates the production of specific antibodies. An antigen is any substance that combines with those specific antibodies.

The term antigen is often used to mean both an antigen and an immunogen.
2) Characteristics : Foreign/Non-self matter

      e.g. Microorganisms, toxins, foreign tissues.  Chemically : Complex molecules: e.g. Protein or polysaccharide

 

 

 

 

 

Hapten/Partial antigen:

Has reactivity (combines with specific antibodies) without immunogenicity (stimulating production of specific antibodies) unless bound to a carrier.
Antigenic Determinant/Epitope

Antibodies are not formed against a whole organism but specific regions or chemical groups.

 

Nature of Antibodies/Immunoglobulins

             Definition: A protein produced by B lymphocytes in response to an immunogen/antigen and is capable of combing with that antigen.

             Gamma Globulins: From separation of blood proteins by electophoresis.

 

3) Antibody structure: Two heavy chains and two light chains.

     Heavy and light chains both have a “C” or constant portion and a “V” or variable portion.

 

V Portion: is different for each kind of Ab and gives the Ab its specificity.

     Ag binding occurs at the V portion.

C Portion: Constant for each class of Ab.

 

Fab: (Antigen binding fragment) The two “arms”

Fc: (Crystallizable fragment) The “trunk” of the antibody.

Contains the complement binding site.

 

Classes of Antibody

IgG: Monomer 80% of serum antibodies

(Most abundant Ab in serum) Can cross the placenta: protects fetus & newborn.

Fixes complement. Enhance phagocytosis; neutralize toxins & viruses.

Half-life = 23 days

 

IgM Pentamer 5-10% of serum antibodies

Fixes complement In blood, lymph, on B cells

Agglutinates microbes; first Ab produced in response to infection. Half-life = 5 days

 

IgA Dimer 10-15% of serum antibodies

Main Ab in mucus secretions, and breast milk.

Mucosal protection. Half-life = 6 days

 

IgD Monomer 0.2% of serum antibodies In blood and lymph.

Receptor on B cells. Half-life = 3 days

 

IgE Monomer 0.002% of serum antibodies

On mast cells and basophils, in blood.

Allergic reactions; lysis of parasitic worms Half-life = 2 days

 

Antibody Response (B Cells)
1o Response: Contact with the Ag for the first time.

     1) Plasma cells ---> Antibodies

     2) Memory cells are formed

 

2o Response/ Anamnestic Response

     Memory cells activated ---> Plasma cells ---> Abs formed quickly and in large numbers.

 

SPECIFIC HOST DEFENSES

    Cell-Mediated Immunity Involves interaction of macrophages and specific T lymphocytes (T-Cells)

 

Pathogens entering the gastrointestinal or respiratory tracts pass through:

Peyer's patches which contain Dendritic cells: act as antigen-presenting cells.

 

 

 

 

T cells differentiate into effector T cells when stimulated by an Ag.

Some effector T cells become memory cells.

 

T Cell Types

 Helper T Cells (CD4, TH)

TH1: Activate cells related to cell-mediated immunity

TH2: Activate B cells to produce  IgM, and IgE

 Cytotoxic T Cells (CD8, TC)

Destroy target cells with perforin.

 

 Delayed Hypersensitivity T Cells (TD)

Associated with allergic reaction, transplant rejection, and tuberculin skin test.

 Suppressor T cells (TS) Turns off immune response when Ag is no longer present.

 

Nonspecific Cells

Activated macrophages: Macrophages stimulated by ingesting Ag or by cytokines.

Natural killer cells: Lymphocytes that destroy virus-infected cells, and tumors.

 

 

T-dependent Antigens

T-independent Antigens

Antibody-Dependent Cell-Mediated Cytotoxicity

 

Self-Tolerance

The immune system does not normally attack self tissues or compounds.

Clonal deletion: During embryonic development lymphocytes with antigen receptors for molecules present in the body are destroyed.

 

Major Histocompatibility Complex (MHC)
Human Leukocyte Antigen Complex (HLA)

Glycoproteins in plasma membrane that enable the immune system to distinguish self from nonself.

Class I MHC: On all nucleated cells.

Class II MHC: On macrophages and B cells.

 

 

 

 




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